References

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Chae, C., 2004. Postweaning multisystemic wasting syndrome: A review of aetiology, diagnosis and pathology. Vet. J., 168: 41-49.
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Dupont, K., E.O. Nielsen, P. Baekbo and L.E. Larsen, 2008. Genomic analysis of PCV2 isolates from Danish archives and a current PMWS case-control study supports a shift in genotypes with time. Vet. Microbiol., 128: 56-64.
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Horlen, K.P., S.S. Dritz, J.C. Nietfeld, S.C. Henry and R.A. Hesse at al., 2008. A field evaluation of pig mortality, performance and infection following commercial vaccination against porcine circovirus type 2. J. Am. Vet. Med. Assoc., 232: 906-912.

Kennedy, S., D. Moffett, F. McNeilly, B. Meehan, J. Ellis, S. Krakowka and G.M. Allan, 2000. Reproduction of lesions of postweaning multisystemic wasting syndrome by infection of conventional pigs with porcine circovirus type 2 alone or in combination with porcine parvovirus. J. Comp. Pathol., 122: 9-24.
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Liu, J., I. Chen and J. Kwang, 2005. Characterization of a previously unidentified viral protein in porcine circovirus type 2-infected cells and its role in virus-induced apoptosis. J. Virol., 79: 8262-8274.
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Misinzo, G., P.L. Delputte, P. Meerts, D.J. Lefebvre and H.J. Nauwynck, 2006. Porcine circovirus 2 uses heparan sulfate and chondroitin sulfate B glycosaminoglycans as receptors for its attachment to host cells. J. Virol., 80: 3487-3494.
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Nawagitgul, P., I. Morozov, S.R. Bolin, P.A. Harms, S.D. Sorden and P.S. Paul, 2000. Open reading frame 2 of porcine circovirus type 2 encodes a major capsid protein. J. Gen. Virol., 81: 2281-2287.
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